Research overview

The published path to RNOVA Tx.

Each step in the RNOVA concept rests on peer-reviewed research from the Kriz laboratory at Université Laval and the CERVO Brain Research Centre.

  1. 2016

    Foundational microglial biology in ALS

    Microglial phenotypes shift adaptively in preclinical stages of SOD1-mediated ALS, with IL-10 controlling early microglial responses.

    Source Gravel et al., J Neurosci, 2016

  2. 2017

    Diverging mRNA and protein networks

    Ribosome profiling of microglia in vivo shows that highly upregulated immune mRNAs are not translated after innate immune challenge.

    Source Boutej et al., Cell Reports, 2017

  3. 2017

    SRSF3-associated translational repression

    Repression is 3′UTR-mediated and involves SRSF3; SRSF3 knockdown increases synthesis of immune proteins in vitro and in vivo.

    Source Boutej et al., Cell Reports, 2017

  4. 2020

    Rethinking immunity in ALS

    Excessive inflammation and inefficient immune responses can coexist in ALS.

    Source Béland et al., Brain Communications, 2020

  5. 2024

    Chronic microglial dysfunction in neurodegeneration

    Chronically activated ALS microglia lose immune functions and acquire an unconventional proteome.

    Source Barreto-Núñez et al., Glia, 2024

  6. 2024

    Experimental SRSF3 targeting

    After experimental stroke, SRSF3-directed siRNA restores translation of selected immune proteins in microglia/macrophages.

    Source Rahimian et al., Molecular Therapy, 2024

  7. Now

    RNOVA Tx translational development

    Development of cell-targeted SRSF3-directed antisense therapeutics, led by ALS.

Advancing a new paradigm in CNS immunotherapy.

RNOVA Tx welcomes conversations with pharmaceutical, biotechnology, scientific and development partners interested in RNA therapeutics and neurodegenerative disease.